PubMed=12219084; DOI=10.1038/nm758
Kelly G.L., Bell A.I., Rickinson A.B.
Epstein-Barr virus-associated Burkitt lymphomagenesis selects for downregulation of the nuclear antigen EBNA2.
Nat. Med. 8:1098-1104(2002)

PubMed=16051863; DOI=10.1128/JVI.79.16.10709-10717.2005; PMCID=PMC1182620
Kelly G.L., Milner A.E., Tierney R.J., Croom-Carter D.S.G., Altmann M., Hammerschmidt W., Bell A.I., Rickinson A.B.
Epstein-Barr virus nuclear antigen 2 (EBNA2) gene deletion is consistently linked with EBNA3A, -3B, and -3C expression in Burkitt's lymphoma cells and with increased resistance to apoptosis.
J. Virol. 79:10709-10717(2005)

PubMed=28960205; DOI=10.1038/cdd.2017.150; PMCID=PMC5762840
Fitzsimmons L., Boyce A.J., Wei W.-B., Chang C., Croom-Carter D.S.G., Tierney R.J., Herold M.J., Bell A.I., Strasser A., Kelly G.L., Rowe M.
Coordinated repression of BIM and PUMA by Epstein-Barr virus latent genes maintains the survival of Burkitt lymphoma cells.
Cell Death Differ. 25:241-254(2018)